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>   首页   >   产品   >   一抗   >   精选抗体   >   F162A Antibody (C-term)   

F162A Antibody (C-term)

Peptide Affinity Purified Rabbit Polyclonal Antibody (Pab)

     
  • 1 - F162A Antibody (C-term) AP10990b
    F162A Antibody (C-term) (Cat. #AP10990b) western blot analysis in mouse bladder tissue lysates (35ug/lane).This demonstrates the F162A antibody detected the F162A protein (arrow).
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Product info
Application
  • Applications Legend:
  • E=ELISA
  • WB=Western Blotting
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin)
  • IP=Immunoprecipitation
  • IF=Immunofluorescence
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • FC=Flow Cytometry
  • DB=Dot Blot
WB, E
Primary Accession Q96A26
Other Accession NP_055182.3
Reactivity Human, Mouse
Host Rabbit
Clonality Polyclonal
Isotype Rabbit Ig
Calculated MW 17342 Da
Additional info
Gene ID 26355
Other Names Protein FAM162A, E2-induced gene 5 protein, Growth and transformation-dependent protein, HGTD-P, FAM162A, C3orf28, E2IG5
Target/Specificity This F162A antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 118-146 amino acids from the C-terminal region of human F162A.
Dilution WB~~1:1000
Format Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification.
StorageMaintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
PrecautionsF162A Antibody (C-term) is for research use only and not for use in diagnostic or therapeutic procedures.
Protein Information
Name FAM162A
Synonyms C3orf28, E2IG5
Function Proposed to be involved in regulation of apoptosis; the exact mechanism may differ between cell types/tissues. May be involved in hypoxia-induced cell death of transformed cells implicating cytochrome C release and caspase activation (such as CASP9) and inducing mitochondrial permeability transition. May be involved in hypoxia-induced cell death of neuronal cells probably by promoting release of AIFM1 from mitochondria to cytoplasm and its translocation to the nucleus; however, the involvement of caspases has been reported conflictingly.
Cellular Location Membrane; Single-pass membrane protein. Mitochondrion
Research Areas

REFERENCES

O'Seaghdha, C.M., et al. Hum. Mol. Genet. 19(21):4296-4303(2010)
Qu, Y., et al. Stroke 40(8):2843-2848(2009)
Cho, Y.E., et al. Hum. Pathol. 40(7):975-981(2009)
Kim, J.Y., et al. FEBS Lett. 580(13):3270-3275(2006)
Lee, M.J., et al. Mol. Cell. Biol. 24(9):3918-3927(2004)

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