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>   首页   >   产品   >   一抗   >   癌症   >   ARSA Antibody (C-term) (Ascites)   

ARSA Antibody (C-term) (Ascites)

Mouse Monoclonal Antibody (Mab)

     
  • 1 - ARSA Antibody (C-term) (Ascites) AM2083a
    ARSA Antibody (C-term) (Cat. #AM2083a) western blot analysis in Jurkat cell line lysates (35μg/lane).This demonstrates the ARSA antibody detected the ARSA protein (arrow).
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Product info
Application
  • Applications Legend:
  • E=ELISA
  • WB=Western Blotting
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin)
  • IP=Immunoprecipitation
  • IF=Immunofluorescence
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • FC=Flow Cytometry
  • DB=Dot Blot
WB, E
Primary Accession P15289
Other Accession NP_000478.2
Reactivity Human
Host Mouse
Clonality Monoclonal
Isotype IgG2a
Clone Names 498CT24.1.2
Calculated MW 53588 Da
Additional info
Gene ID 410
Other Names Arylsulfatase A, ASA, Cerebroside-sulfatase, Arylsulfatase A component B, Arylsulfatase A component C, ARSA
Target/Specificity This ARSA antibody is generated from mice immunized with a KLH conjugated synthetic peptide between 408-439 amino acids from the C-terminal region of human ARSA.
Dilution WB~~1:500~4000
Format Mouse monoclonal antibody supplied in crude ascites with 0.09% (W/V) sodium azide.
StorageMaintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
PrecautionsARSA Antibody (C-term) (Ascites) is for research use only and not for use in diagnostic or therapeutic procedures.
Protein Information
Name ARSA
Function Hydrolyzes cerebroside sulfate.
Cellular Location Endoplasmic reticulum. Lysosome
Research Areas

BACKGROUND

The protein encoded by this gene hydrolyzes cerebroside sulfate to cerebroside and sulfate. Defects in this gene lead to metachromatic leucodystrophy (MLD), a progressive demyelination disease which results in a variety of neurological symptoms and ultimately death. Alternatively spliced transcript variants have been described for this gene.

REFERENCES

Cesani, M., et al. Hum. Mutat. 30 (10), E936-E945 (2009) :
Matzner, U., et al. J. Biol. Chem. 284(14):9372-9381(2009)
Bisgaard, A.M., et al. Clin. Genet. 75(2):175-179(2009)
Lugowska, A., et al. Clin. Genet. 75(1):57-64(2009)
Oshikawa, M., et al. Mol. Vis. 15, 482-494 (2009) :

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