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RAT Camk2a Antibody (ascites)

Mouse Monoclonal Antibody (Mab)

     
  • 1 - RAT Camk2a Antibody (ascites) AM2002a
    RAT Camk2a Antibody (Cat. #AM2002a) western blot analysis in rat brain tissue lysates (35μg/lane).This demonstrates the Camk2a antibody detected the Camk2a protein (arrow).
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Product info
Application
  • Applications Legend:
  • E=ELISA
  • WB=Western Blotting
  • IHC=Immunohistochemistry
  • IHC-P=Immunohistochemistry (Paraffin)
  • IP=Immunoprecipitation
  • IF=Immunofluorescence
  • IC=Immunochemistry
  • ICC=Immunocytochemistry
  • FC=Flow Cytometry
  • DB=Dot Blot
WB, E
Primary Accession P11275
Other Accession P11798, NP_037052.1
Reactivity Rat
Predicted Mouse
Host Mouse
Clonality Monoclonal
Isotype IgM
Clone Names 390CT12.4.4
Calculated MW 54115 Da
Additional info
Gene ID 25400
Other Names Calcium/calmodulin-dependent protein kinase type II subunit alpha, CaM kinase II subunit alpha, CaMK-II subunit alpha, Camk2a
Target/Specificity Purified His-tagged Camk2a protein(Fragment) was used to produced this monoclonal antibody.
Dilution WB~~1:1000~8000
Format Rat monoclonal antibody supplied in crude ascites with 0.09% (W/V) sodium azide.
StorageMaintain refrigerated at 2-8°C for up to 2 weeks. For long term storage store at -20°C in small aliquots to prevent freeze-thaw cycles.
PrecautionsRAT Camk2a Antibody (ascites) is for research use only and not for use in diagnostic or therapeutic procedures.
Protein Information
Name Camk2a
Function CaM-kinase II (CAMK2) is a prominent kinase in the central nervous system that may function in long-term potentiation and neurotransmitter release. Member of the NMDAR signaling complex in excitatory synapses it may regulate NMDAR-dependent potentiation of the AMPAR and synaptic plasticity.
Cellular Location Cell junction, synapse, presynaptic cell membrane. Cell junction, synapse. Note=Postsynaptic lipid rafts
Research Areas

BACKGROUND

CaM-kinase II (CAMK2) is a prominent kinase in the central nervous system that may function in long-term potentiation and neurotransmitter release. Member of the NMDAR signaling complex in excitatory synapses it may regulate NMDAR-dependent potentiation of the AMPAR and synaptic plasticity (By similarity).

REFERENCES

Hund, T.J., et al. J. Clin. Invest. 120(10):3508-3519(2010) Xu, L., et al. Circ. Res. 107(3):398-407(2010) Guetg, N., et al. Proc. Natl. Acad. Sci. U.S.A. 107(31):13924-13929(2010) Blaich, A., et al. Proc. Natl. Acad. Sci. U.S.A. 107(22):10285-10289(2010) Jenkins, M.A., et al. J. Neurosci. 30(15):5125-5135(2010)

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